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雅思閱讀真題及參考答案
無論是在學習還是在工作中,我們最離不開的就是考試真題了,借助考試真題可以更好地考查參試者所掌握的知識和技能。你知道什么樣的考試真題才算得上好考試真題嗎?下面是小編幫大家整理的雅思閱讀真題及參考答案,歡迎閱讀與收藏。
雅思閱讀真題及參考答案 1
1.匹配題(MATCHING TASKS)。如給每段文章配標題。這要求同學們又比較強的總結判斷能力。
2.多重選擇(MALTIPLE-CHOICE TASKS)。如給出A,B,C三個答案, 選擇正確答案。此類題型往往是考細節,所以大家要對本段的細節留心;旧蟻碚f,這類題型的答案在文中會有比較規律平等的排布。
3.辨別正誤(True / FALSE/TASKS)。
該題型還涉及到:(NOT GIVEN / NOT MENTIONED)沒有提到,
有時還會出現下列提法:
ACCURAT / INACCURAT 精確/不精確
SUPPORTED / CONTRADICTED 一致/不一致
CORRECT / INCORRECT 正確與不正確
這類題型相對來說比較難,大家一定要注意忠于原文,跟原文意思完全一致的為正確,相反的為錯誤,原文中沒有給出明確態度的為沒有提到。大家要特別警惕那些跟原文只有一兩個單詞誤差的選項,往往都是陷阱所在。跟原文一致的答案往往通過另一種句式或者措詞表達出來。
4.填空(GAPFILL)。有時要根據文章內容選擇詞或短語填空,有時會給出一些詞或短語進行填空。其實也要根據文章內容來填空,定位的能力非常重要。有時候也需要作出一些判斷確定最后的答案。
5.完成句子(SENTENCE COMPLETION TASKS)。即先給出句子的一部分,未給出部分在文章中找出,或者給出2-3個答案,由你選擇一個。這種題型較難,宜多加練習。
6.完成圖表、示意圖(TABLE、CHART OR DIAGRAM COMPLETION)。 這部分題型是要根據文章所給出的信息,將圖表內缺失的內容(或數據)填補出來,大家需要掌握的是定位的能力,相對來說還是比較容易掌握的'。
7.回答問題(SHORT-ANSWER QUESTION TASKS);卮饐栴}在IELTS閱讀考試中屬于較易回答的題型,通常在考試的前一部分出現。
對于上述幾種雅思閱讀題型,在答題時要采用不同的辦法靈活處理。例如:
在回答完成句子這類題時,就要花一點時間仔細查看文章中的有關部分,根據要求,填寫穩重給出的詞,或者總結出符合要求的答案。
在回答匹配題時,就要在文章中快速找出關鍵詞或短語,并圍繞這些詞或短語附近找出答案。
在回答填空題時,就要通讀文章內容,按順序回答問題。根據文章中所給單詞或文章并利用詞性找出答案,有時候由于字數限制,可能會要求大家稍微改變一下詞性。
雅思閱讀真題及參考答案 2
1. The failure of a high-profile cholesterol drug has thrown a spotlight on the complicated machinery that regulates cholesterol levels. But many researchers remain confident that drugs to boost levels of ’good’ cholesterol are still one of the most promising means to combat spiralling heart disease.
2. Drug company Pfizer announced on 2 December that it was cancelling all clinical trials of torcetrapib, a drug designed to raise heart-protective high-density lipoproteins (HDLs)。 In a trial of 15000 patients, a safety board found that more people died or suffered cardiovascular problems after taking the drug plus a cholesterol-lowering statin than those in a control group who took the statin alone.
3. The news came as a kick in the teeth to many cardiologists because earlier tests in animals and people suggested it would lower rates of cardiovascular disease. There have been no red flags to my knowledge, says John Chapman, a specialist in lipoproteins and atherosclerosis at the National Institute for Health and Medical Research (INSERM) in Paris who has also studied torcetrapib. This cancellation came as a complete shock.
4. Torcetrapib is one of the most advanced of a new breed of drugs designed to raise levels of HDLs, which ferry cholesterol out of artery-clogging plaques to the liver for removal from the body. Specifically, torcetrapib blocks a protein called cholesterol ester transfer protein (CETP), which normally transfers the cholesterol from high-density lipoproteins to low density, plaque-promoting ones. Statins, in contrast, mainly work by lowering the ’bad’ low-density lipoproteins.
Under pressure
5. Researchers are now trying to work out why and how the drug backfired, something that will not become clear until the clinical details are released by Pfizer. One hint lies in evidence from earlier trials that it slightly raises blood pressure in some patients. It was thought that this mild problem would be offset by the heart benefits of the drug. But it is possible that it actually proved fatal in some patients who already suffered high blood pressure. If blood pressure is the explanation, it would actually be good news for drug developers because it suggests that the problems are specific to this compound. Other prototype drugs that are being developed to block CETP work in a slightly different way and might not suffer the same downfall.
6. But it is also possible that the whole idea of blocking CETP is flawed, says Moti Kashyap, who directs atherosclerosis research at the VA Medical Center in Long Beach, California. When HDLs excrete cholesterol in the liver, they actually rely on LDLs for part of this process. So inhibiting CETP, which prevents the transfer of cholesterol from HDL to LDL, might actually cause an abnormal and irreversible accumulation of cholesterol in the body. You’re blocking a physiologic mechanism to eliminate cholesterol and effectively constipating the pathway, says Kashyap.
Going up
7. Most researchers remain confident that elevating high density lipoproteins levels by one means or another is one of the best routes for helping heart disease patients. But HDLs are complex and not entirely understood. One approved drug, called niacin, is known to both raise HDL and reduce cardiovascular risk but also causes an unpleasant sensation of heat and tingling. Researchers are exploring whether they can bypass this side effect and whether niacin can lower disease risk more than statins alone. Scientists are also working on several other means to bump up high-density lipoproteins by, for example, introducing synthetic HDLs. The only thing we know is dead in the water is torcetrapib, not the whole idea of raising HDL, says Michael Miller, director of preventive cardiology at the University of Maryland Medical Center, Baltimore.
(613 words nature)
Questions 1-7
This passage has 7 paragraphs 1-7.
Choose the correct heading for each paragraph from the list of headings below.
Write the correct number i-ix in boxes 1-7 on your answer sheet.
List of Headings
i. How does torcetrapib work?
ii. Contradictory result prior to the current trial
iii. One failure may possibly bring about future success
iv. The failure doesn’t lead to total loss of confidence
v. It is the right route to follow
vi. Why it’s stopped
vii. They may combine and theoretically produce ideal result
viii. What’s wrong with the drug
ix. It might be wrong at the first place
Example answer
Paragraph 1 iv
1. Paragraph 2
2. Paragraph 3
3. Paragraph 4
4. Paragraph 5
5. Paragraph 6
6. Paragraph 7
Questions 7-13
Match torcetrapib,HDLs,statin and CETP with their functions (Questions 8-13)。
Write the correct letter A, B, C or D in boxes 8-13 on your answer sheet.
NB You may use any letter more than once.
7.It has been administered to over 10,000 subjects in a clinical trial.
8.It could help rid human body of cholesterol.
9.Researchers are yet to find more about it.
10. It was used to reduce the level of cholesterol.
11. According to Kashyap, it might lead to unwanted result if it’s blocked.
12. It produced contradictory results in different trials.
13. It could inhibit LDLs.
List of choices
A. Torcetrapic
B. HDLS
C. Statin
D. CETP
(by Zhou Hong)
Suggested Answers and Explanations
1. vi
2. ii
3. vii 本段介紹了torcetrapib和statin的治病原理,但是同時短語in contrast與之前第二段后半段的內容呼應,暗示了這兩種藥在理論上能相輔相成,是理想的搭配。第一個選項無法涵蓋整段意義,故選擇i是錯誤的。
4. iii 本段分析了可能導致torcetrapibl臨床試驗失敗的'原因,后半段指出如果以上推測正確,那么未來的藥物可借鑒這個試驗,設法避免torcetrapib的缺陷,研制出有效的藥物。viii選項無法涵蓋后半段的意思。
5. ix 見首句。
6. v
7. A 見第二段。題目中administer一詞意為用藥,subject一詞為實驗對象之意。
8. B 見第四段… to raise levels of HDLs, which ferry cholesterol out of artery- clogging plaques to the liver for removal from the body.即HDLs的作用最終是將 choleserol清除出人體:… for removal from the body。
9. B 見第四段But HDLs are complex and not entirely understood.
10. C 見第二段… plus a cholesterol-lowering statin,即statin是可以降低cholesterol的。
11. D 見第六段 So inhibiting CETP, … might actually cause an abnormal and irreversible accumulation of cholesterol in the body.
12. A 見第三段。
13. C 見第四段Statins, in contrast, mainly work by lowering the ’bad’ low-density lipoproteins.
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